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Test Code:
PHOSU24

Order Name:
Phosphate (U-24 hrs)

 
Useful For:
Diagnosis and management of a variety of disorders including bone, parathyroid, and renal disease
 
Methodology:
Phosphomolybdate
 
AliasesName:
Phosphate, U-24 hrs.
PO4, U-24 hrs.
Inorganic Phosphorus, U-24 hrs.
Phosphorus, U-24 hrs.
 
 
 
Test Code:
PHOSU24

Order Name:
Phosphate (U-24 hrs)

 
Collection Specimen Or Container:
24-hour urine, Clean containner with preservative
Preservative -  can be selected as list below:
  1. 20 mL of 6N HCL (Preferred) or
  2. 10 mL of 25% HCl or
  3. non preservative if order with Protein (U-24 hrs) or Microalbumin ( U-24 hrs)
Note:
  • Please keep in refrigerated (2-8°C) during collection and transportation
  • 24-hours volume is required.
 
Specimen Testing Type:
24-hour urine,minimum volume 10 mL
 
Sub Mission Container:
Clean container
 
Rejection Criteria:
Wrong preservative type will be reject.
 
Specimen Stabillity:
Specimen Type Temperature Time
24 hours urine Refrigerated, 2oC to 8oC 8 hours
 
 
 
Test Code:
PHOSU24

Order Name:
Phosphate (U-24 hrs)

 
Method detail:
Phosphomolybdate
 
Schedule:
Tested daily (24 hours)
 
Turnaround Time:
Collected specimen to report within 1.5 hours (90 mins)
 
Performing Location:
Chemistry, Laboratory Department Tel. 13224
 
Specimen Retention Time:
7 days
 
 
 
Test Code:
PHOSU24

Order Name:
Phosphate (U-24 hrs)

 
 
Clinical Information:
Approximately 80% of filtered phosphorus is reabsorbed by renal proximal tubule cells. The regulation of urinary phosphorus excretion is principally dependent on regulation of proximal tubule phosphorus reabsorption. A variety of factors influence renal tubular phosphate reabsorption, and consequent urine excretion. Factors which increase urinary phosphorus excretion include high phosphorus diet, parathyroid hormone, extracellular volume expansion, low dietary potassium intake and proximal tubule defects (eg, Fanconi Syndrome, X-linked hypophosphatemic Rickets, tumor-induced osteomalacia). Factors which decrease, or are associated with decreases in, urinary phosphorus excretion include low dietary phosphorus intake, insulin, high dietary potassium intake, and decreased intestinal absorption of phosphorus (eg, phosphate-binding antacids, vitamin D deficiency, malabsorption states).

A renal leak of phosphate has also been implicated as contributing to kidney stone formation in some patients.
 
Reference Value:
0.4 – 1.3 g/day 
 
Interpretation:
Interpretation of urinary phosphorus excretion is dependent upon the clinical situation, and should be interpreted in conjunction with the serum phosphorus concentration.
 
Clinical Reference:
  1. Manufacturer’s reagent package insert, Architect® Phosphorus, Abbott Laboratories, Abbott Park IL 60064 USA, January 2016.
  2. http://www.mayomedicallaboratories.com (Retrieved: 01 Jan 2019)